Benefits of I.V. Vitamin C | Dr. Thomas Levy
While researching thousands of articles over the last few years
in the preparation of my latest book on vitamin C (Levy, 2002),
interesting patterns began to emerge. Even though the effects of
vitamin C on over 25 different infectious diseases and over 100
different toxins were examined, common mechanisms of action became
apparent. This was especially significant to me since I had long
wondered how a single chemical entity (ascorbate, or vitamin C)
could have such dramatically positive clinical effects on such a
wide array of completely unrelated chemical compounds and infectious
agents. Quite literally, there seemed to be no exceptions to this
vitamin C effect. Even if vitamin C did not cure a given infection
or toxic state, it always helped resolve such a condition to some
degree.
Dr. Albert Szent-Gyorgyi, the brilliant scientist who won the Nobel
Prize in 1937 for his discovery of vitamin C, also advanced what
I would call a true theory of life in two of his last publications.
Szent-Gyorgyi (1978, 1980) asserted that energy exchange in the
body can only occur when there is an imbalance of electrons among
different molecules, assuring that electron flow must take place.
Natural electron donators give up electrons to natural electron
acceptors. Szent-Gyorgyi maintained that dead tissue had a full
complement of electrons, a state in which no further exchange or
flow of electrons could take place. Another way of viewing this
is that brisk electron flow and interchange equals health, impaired
or poor electron flow and interchange equals disease, and cessation
of flow and interchange equals death. Vitamin C, as the premier
antioxidant in the body, is perhaps the most important ongoing electron
donor to keep this electron flow at optimal levels.
Oxidation involves the loss of electrons, and an antioxidant counters
this process by supplying electrons. Although vitamin C is the most
important antioxidant in the body, there are many different antioxidants
present in the body, and many of them work to keep the more important
antioxidant substances in the body in the reduced state, which allows
the donation of electrons. For example, vitamin E is an antioxidant
that is fat soluble, which is important in allowing it to be the
primary antioxidant present in the lipid-rich cell membranes of
the body. Vitamin C, which is water soluble, helps to recharge oxidized
vitamin E in those cell membranes back to the electron-rich reduced
form. Even though vitamin C is not the primary antioxidant in the
cell wall, it plays a vital role in maintaining the optimal levels
of the metabolically active antioxidant, vitamin E, at that site.
It appears, then, that the local loss of electrons (oxidation)
represents the primary degeneration, or metabolic breakdown, of
the tissue or chemical substance losing the electrons. An antioxidant
can serve to immediately restore this loss of electrons, resulting
in a prompt "repair" of that acutely oxidized tissue. Also, an antioxidant
can often neutralize the oxidizing agent before it gets a chance
to oxidize, or damage, the tissue initially.
All of the vitamin C/toxin exposure studies reviewed showed one
or more of the following findings or consequences in the test tube,
tissue, intact animal, or human studied:
- 1. Decreased levels of vitamin C and other antioxidants (blood
and/or the tissues most specifically affected)
- 2. Increased levels of oxidative stress in the test setting,
indicating ongoing oxidation
- 3. Increased liver production of vitamin C (in those species
capable of this), as an adaptive response
- 4. Increased rates of consumption of vitamin C and other antioxidants
- 5. A direct correlation between toxin activity and antioxidant
levels (lower antioxidant levels, greater clinical toxicity)
- 6. The acute induction of scurvy or other clinical findings
consistent with the acute depletion of vitamin C
Intravenous Vitamin C with Dr. Tom Levy - >> continued
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